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In a 2023 study, Aryal and Prabhu investigated whether individuals with misophonia exhibit altered late latency auditory potentials, motivated by neuroimaging findings of increased activity in auditory cortical regions and comorbidity with other auditory disorders; they assessed this using standard audiological screening (case history, otoscopy, PTA, HFA, tympanometry) followed by ALLR recording (P1–N1–P2–N2) under controlled conditions with replication. They found distinct severity categories based on A-MISO-S scores, high-pitched sounds as the dominant trigger (~87%), earlier P1 and N1 peak latencies in Misophonic's (contrary to (Schröder et al., 2014)), no reduction in N1 amplitude, no P2/N2 differences, and no hearing threshold differences between groups, attributing discrepancies with prior work to methodological and stimulus differences. The authors conclude that while higher-level auditory processing (P2/N2) appears comparable to controls, earlier P1/N1 latencies suggest possible alterations in early cortical auditory processing in misophonia, supporting ALLR as a potential clinical correlate pending further validation. (Aryal & Prabhu, 2023)

focus on the counter to Schroder

In a 2023 study, Aryal and Prabhu investigated whether individuals with misophonia exhibit altered late latency auditory potentials, using ALLR recording (P1–N1–P2–N2) under controlled conditions with replication. They found distinct severity categories based on A-MISO-S scores, high-pitched sounds as the dominant trigger (~87%), earlier P1 and N1 peak latencies in Misophonic's (contrary to (Schröder et al., 2014)), no reduction in N1 amplitude, no P2/N2 differences, and no hearing threshold differences between groups, attributing discrepancies with prior work to methodological and stimulus differences (Aryal & Prabhu, 2023)